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Dr. Jean-Bernard Durand of M.D. Anderson discussed these cases with Drive at a gathering and suggested they make an effort to determine the cause of this issue, Force said. Gleevec targets three specific proteins, including one called ABL. In chronic myelogenous leukemia, genes referred to as ABL and BCR become fused and create a hybrid BCR-ABL enzyme that is always active. The overactive BCR-ABL, in turn, drives the extreme proliferation of white bloodstream cells that is the hallmark of leukemia. Using viruses that produced for normal ABL and a Gleevec-resistant mutant in laboratory research and in mice, the researchers discovered that Gleevec inhibited the normal enzyme but not the mutant, and the mutant ABL rescued center cells from the toxic effects of Gleevec.